However, Wang et al.[13c] demonstrated that EZH2 directly interacted with MYC in neuroblastoma via MB2, and that ectopically expressed EZH2 has minimal effects on the MYC p‐T58 levels, suggesting that stabilization of MYC by EZH2 occurs via a MYC p‐T58‐independent mechanism. The gene discussed is MYC; the disease is neuroblastoma.