Samimi et al. [15] delved into the mechanism of action of IL-6 while stressing the significant role of inflammation in the pathophysiology of endometriosis; Wenjie et al. [16] further corroborated the importance of IL-6 in the endometriosis pathogenesis through specific research on the expressions of neutrophil to lymphocyte ratio (NLR), C-reactive protein (CRP), IL-6, and IL-17 in endometriosis and their correlations with the disease condition. Here, IL17A is linked to endometriosis.