GPT and metabolic dysfunction-associated steatotic liver disease: Our study showed that patients with MASLD were characterised by a remarkable increase in the oxidation of ApoC‐III proteoforms compared with controls, that this is a MASLD‐specific process, without a major influence exerted by T2DM (Figure 2), and positively correlated to metabolic features of MASLD (BMI, triglycerides HOMA2‐IR, AST and ALT), and negatively correlated with HDL‐C.