Among them, the following genes were found to be downregulated: CXCR4, a mediator of bacterial lipopolysaccharide‐induced inflammatory response; JUNB, involved in interleukin‐1 family signaling pathways; NKG7, essential for cytotoxic degranulation of NK cells and CD8+ T cells and for the activation of CD4+ T cells following infection; RGS1, an inhibitor of B cell chemotaxis; RPS10, responsible for the synthesis of proteins in the cell; and TNFAIP3, an inhibitor of TNF‐mediated apoptosis. The gene discussed is CD8A; the disease is infection.