However, this association must be viewed with caution as this experimental cohort contained more Myf6 lineage tumours with Pax3::Foxo1 knock‐in than Myf6 lineage tumours without Pax3::Foxo1 knock‐in, and thus the prevalence of Myf6 lineage tumours in the ML cluster may be attributable to selection bias. This evidence concerns the gene MYF6 and neoplasm.