Considering our previously proposed role of dAux in a Drosophila PD model, and that the mammalian dAux homolg GAK has been shown to be a potential PD risk factor, we speculate that Atg9 phosphorylation acting downstream of dAux, a glial mechanism identified in the present study, might be important for pathological features implicated in PD. Here, GAK is linked to Parkinson disease.