Some forms of retinitis pigmentosa (RP) in humans arise from this same mutation.[145] Sakai et al.[144] reported consistently greater opsin preservation in bFGF‐NP‐treated retinas, a significantly greater number of photoreceptors, significantly fewer TUNEL‐positive cells, and a significant increase in bFGF levels in treated retinas, verifying that bFGF‐NPs can provide sustained retinal rescue (Figure 11). The gene discussed is FGF2; the disease is retinitis pigmentosa 1.