Xie, Guo, et al. (2017) identified ALB could significantly mitigate early neurovascular dysfunction of subarachnoid hemorrhage rats through suppression of the polarization of macrophages/microglia to the M1 phenotype (interleukin‐1β, CD16, and CD32) and neutrophil invasion (decreased mRNA levels of monocyte chemoattractant protein‐1, cytokine‐induced neutrophil chemoattractant‐1, and CXC chemokine ligand 2). The gene discussed is ALB; the disease is subarachnoid hemorrhage.