In addition, another study in lung adenocarcinoma (LUAD) demonstrated that METTL3 enhances RNA stability and promotes translation of SLC7A11 through m6A-mediated binding of YTHDF1 and YTHDC2, YTHDC2 prefers to bind to m6A-modified SLC7A11 mRNA and promoting its decay [44]. This evidence concerns the gene SLC7A11 and lung adenocarcinoma.