At the same time, different ethnic profiles of AD and chronic forms exhibit distinct immune profiles, with a predominance of Th-1, Th-17, and others, leading to overexpression of various cytokines involved in AD (IL-5, IL-17, IL-12/23, IL-22, IL-9, IL-18, IL-33, IL-25, IL-37) and the release of other immunomodulatory molecules such as thymic stromal lymphopoietin (TSLP) [13,15,16,17]. This evidence concerns the gene IL22 and Alzheimer disease.