As before, the number of adhered AML cells was much less in the presence of anti-CD44, but pFAK was consistently higher in the adhered AML cells (MFI 3133 ± 94) compared to non-adhered (MFI 2052 ± 34) and monoculture (MFI 1661 ± 99) supporting the use of this signaling pathway for adhesion (Figure 5e). This evidence concerns the gene CD44 and acute myeloid leukemia.