We are aware that we tested only interactions between cancer cells, where the cross-talk between elements of the tumour environment could be responsible for these properties—as was shown in the example of cancer-associated fibroblasts (CAFs) in colorectal cancer—causing radioresistance through activation of the TGF-β signalling pathway or transfer miR-93-5p [86,87]. The gene discussed is TGFB1; the disease is neoplasm.