More recent experimental evidence in a mouse model of epidermolysis bullosa acquisita would confirm the immunomodulatory role of vitamin D via increase of CD4+ forkhead box P3 (FoxP3)+ Tregs and B cells (CD19 + IL10+), concomitant with the reduction of proinflammatory Th17 cells [33]. The gene discussed is CD19; the disease is acquired epidermolysis bullosa.