Duchenne muscular dystrophy (DMD) is a severe X-linked hereditary disorder caused by mutations in various regions of the dystrophin gene (DMD) that result in a frameshift and formation of a premature termination codon, leading to either unfunctional truncated protein expression or a complete absence of the full muscular dystrophin isoform (Dp427m) [1]. The gene discussed is DMD; the disease is X-linked disease.