These cells exhibited immunomodulatory abilities, suppressed T-cell proliferation and production of pro-inflammatory cytokines such as TNF-α, IFN-γ, and IL-17, and, in a mouse model, alleviated the symptoms of IBD, as manifested by faster weight recovery, longer bowel length, and improved histological indices of tissue damage [93]. The gene discussed is IL17A; the disease is inflammatory bowel disease.