In our work, we showed that indeed, the percentage of M1-like-type CD80+ peritoneal macrophages was significantly higher in the Muc2−/− mice compared to the C57BL/6 mice, and the percentage of anti-inflammatory M2-like-type CD209+ peritoneal macrophages was significantly lower in the experimental model of IBD. This evidence concerns the gene CD209 and inflammatory bowel disease.