Spontaneous remission occurs in 40% of cases, while 20% progress to kidney failure.[1] Venous thromboembolic (VTE) complications in nephrotic syndrome arise from urinary loss of antithrombin III and reduced protein S activity.[2] Clinically evident VTE events affect 7% of MN patients,[3] and some studies suggest that Anti-PLA2R-associated MN carries a higher VTE risk.[4]. Here, PLA2R1 is linked to nephrotic syndrome.