MBL2 can spike the spike protein of SARS‐CoV‐2 and activate the lectin pathway of complement activation,[9] a process linked to COVID‐19‐associated thrombosis.[10] FCN2, identified as a pathogenic factor, is implicated in thrombocytopenia observed in patients with SLE.[11] CFP, also known as properdin, functions as a positive regulator of the complement cascade by stabilizing C3 and C5 convertases. This evidence concerns the gene CFP and deep vein thrombosis.