In addition, several inflammation‐associated genes, including Retn, Mif, Ptgs1, Ptgs2, Hif1a, and PF4, were upregulated in BM cells from ITP‐exo‐treated mice compared with those from HD‐exo‐treated mice (Figure 1E(iii); Figure S1E, Supporting Information), indicating that ITP‐specific exosomes can be transferred into the BM microenvironment and further impair normal hematopoiesis. The gene discussed is PF4; the disease is autoimmune thrombocytopenic purpura.