The analysis showed that the molecular mechanisms by which E. officinalis treats HCC may be implicated in pathways in cancer, fluid shear stress and atherosclerosis, PI3K-Akt signaling pathway, EGFR tyrosine kinase inhibitor resistance, IL17 signaling and MAPK signaling pathway (Figure 5(Fig. 5)). The gene discussed is AKT1; the disease is atherosclerosis.