In line with the migration data, VSV-S-infected HNSCC cells exhibited elevated expression and secretion levels of CXCL9 and CXCL10 compared to MOCK- or wtVSV-infected cells (Supplementary Fig. S4), suggesting that VSV-S promotes T cell trafficking to tumors not only through releasing PANoptosis-associated pathogen-associated molecular patterns (PAMPs) and/or damage-associated molecular patterns (DAMPs) into the TME, but also by enhancing the production of T cell-attracting chemokines. The gene discussed is CXCL10; the disease is head and neck squamous cell carcinoma.