RAB8A can be activated by RAB11A and plays an essential role in cilia formation and function by interacting with numerous proteins linked to ciliary basal bodies and axons/membranes.[19, 20] Previous studies have shown that primary cilia are indeed absent in prostate cancer and that the development and progression of many tumors may be associated with the absence of primary cilia.[11] We used Ac‐Tubulin and γ‐tubulin as markers to observe primary cilia formation. The gene discussed is RAB11A; the disease is prostate carcinoma.