ARID1A and urinary bladder neoplasm: The sole deletion of ARID1A does not induce bladder tumour development in mice [34], and previous studies have suggested that the absence of ARID1A function alone may not be adequate to initiate tumorigenesis, and that the concurrent loss of TP53 gene function is a pivotal driver for carcinogenesis in urothelial cells [18], indicating an important connection between the cell cycle‐related genes and the SWI/SNF complex.