Given that Ets‐1 is known to be overexpressed in ovarian cancer and has been suggested as a poor prognostic factor [56, 57], it seems reasonable to speculate that the oxidative stress‐mediated antitumor effect, observed after PARP‐1 inhibition, is the result of the transcriptional activity of Ets‐1. This evidence concerns the gene PARP1 and ovarian carcinoma.