Given that Ets‐1 is known to be overexpressed in ovarian cancer and has been suggested as a poor prognostic factor [56, 57], it seems reasonable to speculate that the oxidative stress‐mediated antitumor effect, observed after PARP‐1 inhibition, is the result of the transcriptional activity of Ets‐1. Here, ETS1 is linked to ovarian carcinoma.