IJOQ quantification indicated that infection by Δply Sp resulted in a 35% reduction in E‐cadherin organization (Figure 2a, “Priming: Δply”), consistent with the moderate effects of infection of immortalized monolayers by Δply Sp and indicative of a PLY‐independent pathway for AJC disruption (Xu et al. 2023). The gene discussed is CDH1; the disease is infection.