Treatment with CDDO indeed enhanced E‐cadherin organization in uninfected young mouse ALI monolayers (Figure 5a,b, “Young”: “CDDO” vs. “Veh.”) and was associated with resistance to E‐cadherin dissolution upon Sp infection (Figure 5a,b, “Young”: “Sp + CDDO” vs. “Sp + Veh.”). This evidence concerns the gene CDH1 and infection.