Recent studies have reported that transforming growth factor-β1 (TGF-β1) can give rise to endothelial-to-mesenchymal transition (EndoMT) [12], which means that the endotheliocytes loss their endothelial features and some characteristics of mesenchymal cells or fibroblasts emerge, further leading to the occurrence of atherosclerosis or cardiac fibrosis to aggravate the adverse left ventricular remodeling post-MI [13, 14]. Here, TGFB1 is linked to myocardial infarction.