Replacement of Ahl with the wild‐type from the CBA/CaJ strain successfully prevents accelerated age‐related hearing loss.[66, 67] So far, whether AAVR overexpression could enhance the infection efficiency of other viruses is not clear, nor is whether overexpression of AAVR may increase infection risk from other pathogens beyond mediating AAV endocytosis. The gene discussed is KIAA0319L; the disease is infection.