TRMT1 and viral infectious disease: SARS-CoV-2 employs multiple mechanisms to disrupt host protein synthesis (Thoms et al., 2020; Finkel et al., 2021; Hsu et al., 2021; Puray-Chavez et al., 2022; Eriani and Martin, 2022; Zhang et al., 2022; Banerjee et al., 2020), and Mpro-mediated cleavage of human TRMT1 could contribute to the modulation of cellular translation during viral infection.