This is further substantiated by literature indicating the role of IL-17 a cytokine in promoting sociability in mouse models of neurodevelopmental disorders.92 The decreased ratio of Th17/Treg cells in the spleens of ASD hFMT mice, therefore, may have contributed to the observed behavioral deficits, reinforcing the significance of the gut-immune-brain axis in ASD. The gene discussed is IL17A; the disease is neurodevelopmental disorder.