PLA2G4A and pulmonary arterial hypertension: The links between GJIC and inflammatory mediator responses to LMW PAHs (1-MeA and Flthn) were also previously determined by our group using inhibitors of P38 MAPK, cPLA2, and other anti-inflammatory inhibitors, where these inhibitors prevented the LMW PAH-induced dysregulation of GJIC in vitro [29,30].