Moreover, the pro-autophagic actions of DPG and activation of the AMP-activated protein kinase (AMPK)/mechanistic target of rapamycin (mTOR) pathway have been characterized to curb rodent models of lipopolysaccharide-evoked Alzheimer-like manifestations [30], colitis [25], hepatic steatosis [31], and pancreatic injury [32]. The gene discussed is MTOR; the disease is steatosis.