Liu has identified that CA, DCA, and CDCA significantly activate the FXR receptor, resulting in the increased expression of BTB and CNC homology 1 (BACH1), glutathione (GSH) synthetase, and GPX4 in gastric cancer cells of HGC-27 and MKN-45 in a ferroptosis-dependent manner [106]. The gene discussed is GPX4; the disease is gastric cancer.