A study that investigated the impact of GLP-1 receptor agonists on infectious (influenza) and non-infectious (bleomycin-induced) lung inflammation in wild-type and GLP-1 receptor knockout mice revealed that in the absence of the GLP-1 receptor, bleomycin-induced lung injury was less severe, while activation of the GLP-1 receptor was correlated with increased pulmonary inflammation mediated through the sympathetic nervous system. The gene discussed is GLP1R; the disease is influenza.