Furthermore, exceedance of its commonly used cut-off (0.6 mV) was associated with statistically significantly more severe CTEPH (higher mPAP, PVR, and NT-pro-BNP levels and lower tricuspid annular plane systolic excursion (TAPSE)) and a statistically insignificantly increased risk of death and higher troponin levels in a previous study [8]. Here, NPPB is linked to chronic thromboembolic pulmonary hypertension.