Given the expression of BCL2A1 in neutrophils infiltrating the EPM [17], it is likely that the intensity of BCL2A1 expression, as measured using QuPath, increases with the stage progression of acute HCA in the EPM and correlates positively with the severity of IAI, as indicated by the amniotic fluid (AF) WBC count. This evidence concerns the gene BCL2A1 and hepatocellular adenoma.