CXCL13 is produced by CD68+ and CD206+ alveolar macrophages from patients with IPF, and TNF-α and IL-10 control optimal CXCL13 gene expression in alveolar macrophages, activating the NF-κB and JAK/STAT pathways [326]. Here, CXCL13 is linked to idiopathic pulmonary fibrosis.