Furthermore, several urinary biomarkers, such as urinary NGAL (uNGAL), KIM-1, chemokine C-C-motif ligand 14 (CCL-14), soluble triggering receptor expressed by myeloid cells (sTREM), tissue inhibitor of metalloproteinases-2 insulin-like growth factor-binding protein 7 (TIMP-2) (IGFBP7), IL-18, CCL-2, and liver-type fatty acid binding protein (L-FABP), have all been proposed as early markers for SA-AKI [46,56,57,58,59]. This evidence concerns the gene CCL2 and acute kidney injury.