Lai et al. discovered that delivering exosomal miR-193b-3p to mice with subarachnoid hemorrhage (SAH) ameliorated subsequent neuroinflammatory responses and neurobehavioral impairments, presumably by downregulating histone deacetylase 3 (HDAC3) expression and upregulating NF-κB p65 acetylation [93]. This evidence concerns the gene HDAC3 and subarachnoid hemorrhage.