TCGA-GBM exhibits upregulations in AR and S1PR3 (Sphingosine-1-Phosphate Receptor 3), both at 5.5-fold, and downregulations in HTR1A (−186.3-fold) and HTR3B (5-Hydroxytryptamine Receptor 3B) (−95.4-fold). This evidence concerns the gene S1PR3 and glioblastoma.