These data provide the first demonstration of the correlations between behavioral scores and the biochemical alterations in p62 and/or GLAST expression as a result of a prenatal immune challenge, which may support the hypothesis that prenatal infection disrupts processing within the prefrontal cortex, consistent with findings regarding the pathophysiology of neuropsychiatric disorders, including schizophrenia and/or autism [56,57]. The gene discussed is SLC1A3; the disease is schizophrenia.