Reciprocally, we did not observe the expected increase in tumour number upon >crol-RNAi (Fig. 6D, E), which is probably attributed to already strong mitotic stimuli such as EGF signalling acting on Stg and Cyclins during tumorigenesis outweighing reduced antiproliferative Crol effects28,50. Here, C6orf15 is linked to neoplasm.