The transient resistance of cancer cells to the drug oxaliplatin is the result of drug-induced upregulation of the BLACAT1 lncRNA that, in turn, serves as an inhibitory sponge (ceRNA) of miR-136 leading to elevated expression of ABCB1 and export of the drug from the cancer cells (Wu et al. 2018). The gene discussed is BLACAT1; the disease is cancer.