Additionally, two subclusters (Neu_3, Neu_4) exhibited high levels of IFN‐stimulated gene expression (Figure 5l), likely resulting from the interaction with IFN‐γ+ cells.[20] These findings suggest that the decreased infiltration of tumor‐promoting neutrophils may account for tumor growth inhibition in mRNA‐treated groups. The gene discussed is IFNA1; the disease is neoplasm.