The elevated miR-214-3p had the potential to hasten FAP fibrogenesis by adjusting the FGF2/FGFR1/TGF-β axis, which shed light on new strategies for the treatment of fibrous degeneration of Duchenne muscular dystrophy (DMD) by interfering miR-214-3p.59 This evidence concerns the gene FAP and Duchenne muscular dystrophy.