SLC7A5 and autism: More recently, it has been reported that SLC7A5‐mediated amino acid transport is crucial for neuronal excitability and survival,[61] in line with a previous study showing that SLC7A5 mutations cause autism via the disruption of normal levels of brain branch chain amino acids (BCAAs) in humans,[62] suggesting a key role of SLC7A5 in neuronal viability and function.