SLC3A2 and neoplasm: We also found that c‐Myc overexpression elevated SLC1A5/SLC3A2/SLC7A5 expression in RTN4IP1‐deficient cells, and promoted cell proliferation as well as tumor growth upon RTN4IP1 knockdown, demonstrating the essential role of SLC1A5/SLC3A2/SLC7A5 in RTN4IP1‐mediated cell proliferation (Figure S7F–J, Supporting Information).