AAV‐Dexras1‐shRNA‐GFP effectively infected the peri‐infarct cortex (Figure 2B), robustly decreased the levels of Dexras1 and SNO‐Dexras1(Figure 2C,D), and significantly ameliorated stroke‐induced impairment of motor function 11–46 d after stroke (Figure 2E–G). The gene discussed is RASD1; the disease is Stroke.