BCL2 and Alzheimer disease: The male‐specific enrichment in apoptosis‐related pathways aligns with studies showing the activity of the pro‐apoptotic protein BCL2‐associated death promoter (BAD) is significantly elevated in neurons of male AD models, while the female‐specific alterations in Wnt signaling could contribute to the reported differences in synaptic plasticity and neurogenesis between sexes in AD.69, 70