Similarly, in another AE-IPF mouse model with BLM and Haemophilus influenzae, IL-17 knockout mice with AE-IPF had quicker body weight recovery, milder pulmonary inflammation and fibrosis, and weaker neutrophil and eosinophil responses than wild-type mice with AE-IPF (103). The gene discussed is IL17A; the disease is idiopathic pulmonary fibrosis.