Our investigation unveiled significant connections between HNRNPC levels and multiple pathways implicated in tumor progression, including angiogenesis, apoptosis, cellular response to hypoxia, extracellular matrix (ECM) degradation, DNA repair, DNA replication, epithelial-mesenchymal transition (EMT), ferroptosis, G2/M checkpoint, IL-10 anti-inflammatory response, immune response, and tumor proliferation (Fig. 9G–R). This evidence concerns the gene IL10 and neoplasm.