Consistent with these previous findings, our study demonstrated that non‐BM‐derived CD34+ cells can create an activated fibrotic microenvironment to promote the repair of damaged/disrupted adventitial connective tissues induced by AAA pathological conditions and may enhance the biomechanical strength of aneurysmal aortas, thereby limiting AAA progression and protecting them from rupture. Here, CD34 is linked to triple-A syndrome.