In support of the viral mechanisms by which HLA class I molecules could lead to MG are the results of an experimental study showing that the HLA-B*18:01 allele, with a slightly higher frequency among our myasthenic patients (10% vs. 8.75%), possesses the ability to present an autoantigen homologous to the epitope of Epstein Bar virus (EBV) to the T cytotoxic cells [36]. This evidence concerns the gene HLA-B and myasthenia gravis.